Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent

Takanari, H., Bourgonje, V.J., Fontes, M.S.C., Raaijmakers, A., Driessen, H.E., Jansen, J.A., Nagel, R.V.D., Kok, B., Stuijvenberg, L.V., Boulaksil, M., Takemoto, Y., Yamazaki, M., Tsuji, Y., Honjo, H., Kamiya, K., Kodama, I., Anderson, M.E., Heyden, M.A.G.V.D., Rijen, H.V.V., &amp; Veen, T.A.V. (2016). Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent. <em>Cardiovascular Research</em>. https://doi.org/10.1093/cvr/cvw173

Takanari H, Bourgonje VJ, Fontes MSC, Raaijmakers A, Driessen HE, Jansen JA, et al. Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent. Cardiovascular Research. 2016. doi:10.1093/cvr/cvw173.

@article{hiroki2016Calmod, title = {Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent}, author = {Hiroki Takanari and Vincent J.A. Bourgonje and Magda S. C. Fontes and A. Raaijmakers and Helen E. Driessen and John A. Jansen and Roel van der Nagel and Bart Kok and Leonie van Stuijvenberg and M. Boulaksil and Yoshio Takemoto and Masatoshi Yamazaki and Yukiomi Tsuji and Haruo Honjo and Kaichiro Kamiya and Itsuo Kodama and Mark E. Anderson and Marcel A. G. van der Heyden and Harold V.M. van Rijen and Toon A.B. van Veen and Marc A. Vos}, journal = {Cardiovascular Research}, year = {2016}, doi = {10.1093/cvr/cvw173}, }