Open access · US-GOV via ClinicalTrials.gov Clinical trial

Autophagy Maintains Vascular Function Through a Novel Glycolysis-linked Pathway Regulating eNOS

Authors not listed

University of Utah · 2018

Disabled macroautophagy Human Randomized controlled trial

Abstract

Aging is inevitable and is the primary risk factor for developing cardiovascular disease. The molecular mechanisms that drive vascular dysfunction in the context of aging are incompletely understood. The overall hypothesis is that the age-related decline in endothelial cell (EC) autophagy leads to arterial dysfunction. This study will determine whether physiological shear-stress affects autophagosome formation and nitrous oxide (NO) generation in ECs.

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Source: ClinicalTrials.gov
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Fetched: 2026-10-21 MST

Cite this

APA

Anonymous. (2018). Autophagy Maintains Vascular Function Through a Novel Glycolysis-linked Pathway Regulating eNOS. <em>University of Utah</em>. https://clinicaltrials.gov/study/NCT04200560

Vancouver

Anonymous. Autophagy Maintains Vascular Function Through a Novel Glycolysis-linked Pathway Regulating eNOS. University of Utah. 2018.

BibTeX

@misc{anon2018Autoph, title = {Autophagy Maintains Vascular Function Through a Novel Glycolysis-linked Pathway Regulating eNOS}, author = {Anonymous}, journal = {University of Utah}, year = {2018}, }