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A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy

Clemens A. Schmitt, Jordan S. Fridman, Meng Yang, Soyoung Lee, Eugene Baranov, Robert M. Hoffman, Scott W. Lowe

Cell · 2002 · ▲ 1,037 citations

Cellular senescence

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Provenance

Source: OpenAlex
DOI: 10.1016/s0092-8674(02)00734-1
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Fetched: 2026-05-31 MST

Cite this

APA

Schmitt, C.A., Fridman, J.S., Yang, M., Lee, S., Baranov, E., Hoffman, R.M., & Lowe, S.W. (2002). A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy. <em>Cell</em>. https://doi.org/10.1016/s0092-8674(02)00734-1

Vancouver

Schmitt CA, Fridman JS, Yang M, Lee S, Baranov E, Hoffman RM, et al. A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy. Cell. 2002. doi:10.1016/s0092-8674(02)00734-1.

BibTeX

@article{clemens2002ASenes, title = {A Senescence Program Controlled by p53 and p16INK4a Contributes to the Outcome of Cancer Therapy}, author = {Clemens A. Schmitt and Jordan S. Fridman and Meng Yang and Soyoung Lee and Eugene Baranov and Robert M. Hoffman and Scott W. Lowe}, journal = {Cell}, year = {2002}, doi = {10.1016/s0092-8674(02)00734-1}, }