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Evidence brief · hallmark

Chronic inflammation

Synthesized from 10 indexed sources · 2026-06-03 MST · preview (no AI key)
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Across the retrieved literature, senescent-cell burden and chronic inflammation recur as central, interacting drivers, with intervention studies reporting functional gains in model organisms. Key works: [1][2][3][4][5].

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Sources

  1. [1] The Occurrence of Sleep-Disordered Breathing among Middle-Aged Adults
  2. [2] Hepatocellular carcinoma
  3. [3] Multi-ethnic reference values for spirometry for the 3–95-yr age range: the global lung function 2012 equations
  4. [4] Inflamm‐aging: An Evolutionary Perspective on Immunosenescence
  5. [5] The Senescence-Associated Secretory Phenotype: The Dark Side of Tumor Suppression
  6. [6] Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor
  7. [7] Ferroptosis: molecular mechanisms and health implications
  8. [8] WHO Child Growth Standards based on length/height, weight and age
  9. [9] NRF2 as a guardian of health: from an ancient survival pathway to a modern therapeutic target.
  10. [10] Telomerase and chronic inflammation as central molecular links in aging
All Chronic inflammation works →