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Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis

Hyun Jik Lee, Young Hyun Jung, Gee Euhn Choi, Jun Sung Kim, Chang Woo Chae, Jae Ryong Lim, Seo Yihl Kim, Jee Hyeon Yoon, Ji Hyeon Cho, Sei‐Jung Lee, Ho Jae Han

Cell Death and Differentiation · 2020 · ▲ 179 citations

Mitochondrial dysfunction

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Provenance

Source: OpenAlex
DOI: 10.1038/s41418-020-0593-1
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Fetched: 2026-06-24 MST

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APA

Lee, H.J., Jung, Y.H., Choi, G.E., Kim, J.S., Chae, C.W., Lim, J.R., Kim, S.Y., Yoon, J.H., Cho, J.H., Lee, S., & Han, H.J. (2020). Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis. <em>Cell Death and Differentiation</em>. https://doi.org/10.1038/s41418-020-0593-1

Vancouver

Lee HJ, Jung YH, Choi GE, Kim JS, Chae CW, Lim JR, et al. Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis. Cell Death and Differentiation. 2020. doi:10.1038/s41418-020-0593-1.

BibTeX

@unpublished{hyun2020Urolit, title = {Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis}, author = {Hyun Jik Lee and Young Hyun Jung and Gee Euhn Choi and Jun Sung Kim and Chang Woo Chae and Jae Ryong Lim and Seo Yihl Kim and Jee Hyeon Yoon and Ji Hyeon Cho and Sei‐Jung Lee and Ho Jae Han}, journal = {Cell Death and Differentiation}, year = {2020}, doi = {10.1038/s41418-020-0593-1}, }