Skip to content
AgingNexus
☰ Menu

Evidence brief · hallmark

Epigenetic alterations

Synthesized from 10 indexed sources · 2026-06-03 MST · preview (no AI key)
Preview synthesis (deterministic, no AI key configured). Set a provider key to generate the full brief.

Synthesis (preview — no live AI key for the active provider):

Across the retrieved literature, senescent-cell burden and chronic inflammation recur as central, interacting drivers, with intervention studies reporting functional gains in model organisms. Key works: [1][2][3][4][5].

Set the active provider's API key to generate full model-written analysis.

Sources

  1. [1] Modernity and Self-Identity: Self and Society in the Late Modern Age
  2. [2] Integrated genomic analyses of ovarian carcinoma
  3. [3] Global, regional, and national age–sex specific all-cause and cause-specific mortality for 240 causes of death, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013
  4. [4] DNA methylation age of human tissues and cell types
  5. [5] DNA methylation patterns and epigenetic memory
  6. [6] Integrative analysis of 111 reference human epigenomes
  7. [7] Comprehensive molecular characterization of gastric adenocarcinoma
  8. [8] Body fat assessed from total body density and its estimation from skinfold thickness: measurements on 481 men and women aged from 16 to 72 Years
  9. [9] NRF2 at the crossroads of Parkinson’s disease and aging: Mechanistic insights and translational perspectives
  10. [10] Telomerase and chronic inflammation as central molecular links in aging
All Epigenetic alterations works →