Alessandro Bitto

PLoS ONE · 2012

⊕ Open access · CC-BY

Astrocyte Senescence as a Component of Alzheimer’s Disease

▲ 581

eLife · 2016

⊕ Open access · CC-BY

Transient rapamycin treatment can increase lifespan and healthspan in middle-aged mice

▲ 476

Cold Spring Harbor Perspectives in Medicine · 2015

⊕ Preprint · OA

Biochemical Genetic Pathways that Modulate Aging in Multiple Species: Figure 1.

▲ 121

Aging Cell · 2021

⊕ Open access · CC-BY

17‐a‐estradiol late in life extends lifespan in aging UM‐HET3 male mice; nicotinamide riboside and three other drugs do not affect lifespan in either sex

▲ 118

Frontiers in Genetics · 2015

⊕ Open access · CC-BY

Dose-dependent effects of mTOR inhibition on weight and mitochondrial disease in mice

▲ 94

Aging Cell · 2020

⊕ Open access · CC-BY

SS‐31 and NMN: Two paths to improve metabolism and function in aged hearts

▲ 61

Kidney International · 2018

⊕ Open access · OA

mTOR inhibitors may benefit kidney transplant recipients with mitochondrial diseases

▲ 59

Cell Metabolism · 2014

⊕ Open access · OA

Rejuvenation: It’s in Our Blood

▲ 27

Nature Metabolism · 2023

⊕ Preprint · OA

Acarbose suppresses symptoms of mitochondrial disease in a mouse model of Leigh syndrome

▲ 20

Innovation in Aging · 2019

⊕ Open access · CC-BY

AGING AND MITOCHONDRIAL DISEASE: SHARED MECHANISMS AND THERAPIES?

▲ 2

Innovation in Aging · 2020

⊕ Open access · CC-BY

Acarbose suppresses symptoms of mitochondrial disease in a mouse model of Leigh Syndrome

▲ 1

bioRxiv (Cold Spring Harbor Laboratory) · 2022

⊕ Preprint · CC-BY

Acarbose suppresses symptoms of mitochondrial disease in a mouse model of Leigh Syndrome

▲ 0